Lactate Production During Inspiratory Threshold Loading in COPD

Tom van der Touw; G Sturdy; S Jenkins; D Hillman; P Eastwood

doi:10.1111/j.1440-1843.2003.supp_1.x

Abstract

The anaerobic threshold has been used as a marker to identify optimal work load for whole body endurance training. To date there has been no convincing evidence of significant anaerobic metabolism during inspiratory muscle training in COPD. Its absence suggests (a) training loads may have been inadequate; or (b) inspiratory muscles produce insufficient lactate to be detected. We examined the relationship between inspiratory load and arterial blood lactate concentration ([La]) during progressive inspiratory threshold loading (PTL) before and after an 8 week program of high intensity inspiratory threshold training in seven COPD patients (FEV1<53% predicted). During PTL, load was increased by 10cm H2O/minute until exhaustion. [La] was measured at the end of each load via a radial arterial catheter.

Original language	English
Pages	A45-A45
DOIs	https://doi.org/10.1111/j.1440-1843.2003.supp_1.x
Publication status	Published - 2003
Event	TSANZ ASM 2003: Thoracic Society of Australia and New Zealand Annual Scientific Meeting - Adelaide, Australia Duration: 4 Apr 2003 → 9 Apr 2003

Conference

Conference	TSANZ ASM 2003: Thoracic Society of Australia and New Zealand Annual Scientific Meeting
City	Adelaide, Australia
Period	4/04/03 → 9/04/03

Keywords

Respiratory Diseases

Access to Document

10.1111/j.1440-1843.2003.supp_1.x

https://hdl.handle.net/1959.11/10062

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Cite this

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title = "Lactate Production During Inspiratory Threshold Loading in COPD",

abstract = "The anaerobic threshold has been used as a marker to identify optimal work load for whole body endurance training. To date there has been no convincing evidence of significant anaerobic metabolism during inspiratory muscle training in COPD. Its absence suggests (a) training loads may have been inadequate; or (b) inspiratory muscles produce insufficient lactate to be detected. We examined the relationship between inspiratory load and arterial blood lactate concentration ([La]) during progressive inspiratory threshold loading (PTL) before and after an 8 week program of high intensity inspiratory threshold training in seven COPD patients (FEV1<53\% predicted). During PTL, load was increased by 10cm H2O/minute until exhaustion. [La] was measured at the end of each load via a radial arterial catheter.",

keywords = "Respiratory Diseases",

author = "\{van der Touw\}, Tom and G Sturdy and S Jenkins and D Hillman and P Eastwood",

year = "2003",

doi = "10.1111/j.1440-1843.2003.supp\_1.x",

language = "English",

pages = "A45--A45",

note = "TSANZ ASM 2003: Thoracic Society of Australia and New Zealand Annual Scientific Meeting ; Conference date: 04-04-2003 Through 09-04-2003",

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TY - CONF

T1 - Lactate Production During Inspiratory Threshold Loading in COPD

AU - van der Touw, Tom

AU - Sturdy, G

AU - Jenkins, S

AU - Hillman, D

AU - Eastwood, P

PY - 2003

Y1 - 2003

N2 - The anaerobic threshold has been used as a marker to identify optimal work load for whole body endurance training. To date there has been no convincing evidence of significant anaerobic metabolism during inspiratory muscle training in COPD. Its absence suggests (a) training loads may have been inadequate; or (b) inspiratory muscles produce insufficient lactate to be detected. We examined the relationship between inspiratory load and arterial blood lactate concentration ([La]) during progressive inspiratory threshold loading (PTL) before and after an 8 week program of high intensity inspiratory threshold training in seven COPD patients (FEV1<53% predicted). During PTL, load was increased by 10cm H2O/minute until exhaustion. [La] was measured at the end of each load via a radial arterial catheter.

AB - The anaerobic threshold has been used as a marker to identify optimal work load for whole body endurance training. To date there has been no convincing evidence of significant anaerobic metabolism during inspiratory muscle training in COPD. Its absence suggests (a) training loads may have been inadequate; or (b) inspiratory muscles produce insufficient lactate to be detected. We examined the relationship between inspiratory load and arterial blood lactate concentration ([La]) during progressive inspiratory threshold loading (PTL) before and after an 8 week program of high intensity inspiratory threshold training in seven COPD patients (FEV1<53% predicted). During PTL, load was increased by 10cm H2O/minute until exhaustion. [La] was measured at the end of each load via a radial arterial catheter.

KW - Respiratory Diseases

U2 - 10.1111/j.1440-1843.2003.supp_1.x

DO - 10.1111/j.1440-1843.2003.supp_1.x

M3 - Abstract

SP - A45-A45

T2 - TSANZ ASM 2003: Thoracic Society of Australia and New Zealand Annual Scientific Meeting

Y2 - 4 April 2003 through 9 April 2003

ER -